Greek ophthalmologist

It is believed that Behcet's disorder may be an auto insubordinate disease. Triolo et al (459) found spunky gamma interferon, just not interleukin 4, levels in the supernatants of lymphocytes from patients with active Behcet's disease cultured in the presence of cow's draw proteins. Levels equaled those obtained from cultures stimulated with PHA. Patients with active Behcet's disease also exhibited higher levels of anti-?-casein and anti-?-lactoglobulin IgG and IgA antibodies. These results indicate an active immune response occurs in Behcet's disease with an increased frequency of antibodies to cow's milk protein

There is an increasing awareness that consumption of cow's milk previous(predicate) in life may constitute a lay on the line factor for autoimmune disease such as sixfold sclerosis, mild rheumatoid arthritis in rabbits, and type 1 diabetes, and may also be a factor in the aetiology of Behcet's disease (Triolo et al 459). The researchers suggest that cow's milk components may keep up detrimental effects on the gut or on systemic immunoreactivity leading to a disturbed peripheral margin mechanism or enhanced susceptibility to viral infections (461). A diet rich in cow's milk may domiciliate antigens that elicit an immune response that cross reacts with self, overcoming self tolerance. extravaganza of the peptides could be available through intestinal flora, such as the heat shock proteins that have high relevance in the pathogenesis o

Vascular manifestations are the study classical features of Behcet's disease, and phlebothrombosis may affect all move of the body, including the dural sinus (Wechsler and Piette 1199). Damage to arteries, seen in five per centum to 35 percent of patients, can cause aneurysms and arterial thrombosis, which hunt a poor prognosis because the aneurysms frequently rupture, particularly those in the pulmonary vessels. In the heart, cardiac lesions include thickening and intraventricular thrombosis. Vasculitis of the coronary arteries can cause infarctions or aneurysms, and often requires operative treatment.

Al-Araji, A., Sharquie, K., & Al-Rawi, Z. "Prevalence and patterns of neurological involvement in Behcet's disease: a prospective study from Iraq.

" Journal of Neurology, Neurosurgery and Psychiatry. 74(5): 608-613.

Wechsler, B., & Piette, J. C. "Behcet's disease: retains most of its mysteries." British Medical Journal, 304(6836):1199-1200.

Several different treatments have been tried for Behcet's disease, including colchicine, corticosteroids, dapsone, thalidomide, levamisole, immunosuppressive drug agents, immunoglobulins, penicillin, and plasmapheresis (Wechsler and Piette 1200). Short-term results with azathioprine and cyclosporin A for the optic problems in Behcet's disease seem promising. High-dose intravenous steroid therapy has proved safe and rough-and-ready for the treatment of severe, vision-threatening acute posterior segment uveitis attacks and for improving visual function in these patients (Toker, Kazokoglu and Acar 521). It has proved effective in optic vasculitis, papillitis, and retinitis. Some success has been reported in the disease utilise subcutaneous injections of recombinant human interferon ?-2a for mucocutaneous lesions, uveitis, and arthritis in patients resistant to high dose corticosteroids (Stanford 381; Calguneri et al 492). These latter researchers evaluated its efficacy, toxicity, and tolerability in the management of Beh
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